Hfq, Crc, and antibiotic resistance in P. aeruginosa
This study examines carbon catabolite repression and its indirect effects on antibiotic susceptibility in Pseudomonas aeruginosa.
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How Pseudomonas aeruginosa responds to colistin and tobramycin
This study combines RNA-seq and ribosome profiling to show how Pseudomonas aeruginosa rewires both transcription and translation when challenged with the last-resort antibiotics colistin and tobramycin.
Hfq regulates carbapenem susceptibility in Pseudomonas aeruginosa
This paper shows that the RNA chaperone Hfq controls carbapenem susceptibility in Pseudomonas aeruginosa through two different post-transcriptional routes acting on the porins OprD and OpdP.
Metabolic control of Hfq-dependent antibiotic susceptibility in Pseudomonas aeruginosa
This paper asks whether the Hfq/Crc/CrcZ metabolic control system can be used to make Pseudomonas aeruginosa more sensitive to antibiotics, and shows that the answer is yes.
How Crc modulates Hfq-dependent RNA regulation in Pseudomonas aeruginosa
This paper explains how the catabolite repression protein Crc modulates Hfq-dependent translational control in Pseudomonas aeruginosa by stabilizing Hfq/RNA assemblies and competing with sRNA access to Hfq.
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